Health & Fitness

A new study finds that whilst climate anxiety is low amongst the UK public, it may be an important driver of climate action such as cutting down on waste.

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Researchers have found that dual bronchodilators -- long-lasting inhalers that relax the airways and make it easier to breathe -- do little to help people who do not have chronic obstructive pulmonary disease (COPD), but who do have respiratory symptoms and a history of smoking.

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Though many of us may seek a quiet place in which to study, 'noise' may play a key role in helping some people improve their learning potential.

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Enzymes are proteins composed of individual amino acids. They are necessary to speed up many cellular functions and biological processes. Your body secretes enzymes to catalyze biological reactions, making them vital to good health and longevity.¹

Each enzyme has a different function, for example, superoxide dismutase, glutathione peroxidase and catalase have antioxidant functions.² Digestive enzymes such as protease, lipase and amylase are needed for digestion and nutrient absorption and elimination,³ while molecular motor enzymes such as myosin and actin are needed for the activation of muscle contractions.⁴

The featured lecture reviews the fundamentals of what enzymes are, the main types of enzymes found in your body and in supplements, how enzymes are affected by environmental factors such as your body's pH and temperature, and why they're so important for health.

Enzymes 101

An enzyme's shape is an important key to understanding the benefits of enzyme therapy, because the shape of the protein determines its function. You could liken enzymes to specialized keys cut to fit specific locks, with the locks in this case being biochemical reactions.

Considering the tens of thousands of biochemical reactions occurring in your body at any given time, it stands to reason there are tens of thousands of enzymes. An interesting feature of enzymes is that while they catalyze biochemical reactions, they're not used up in the reaction. They merely assist and accelerate reactions.

By lowering the amount of energy needed for a reaction to occur, they allow for reactions that otherwise would not be possible, or would be too slow to keep up with your body's demands. This is also why enzyme deficiencies are thought to contribute to more rapid aging.

Types of Enzymes and Their Functions

Enzymes can be broadly divided into the following categories:⁵

3 Main Types of Enzyme Supplements

Enzymes found in enzyme supplements used for enzyme therapy are known as hydrolases. As the name implies, they use a water molecule to cut certain bonds along the amino acid chain. Supplemental enzymes can be divided into three basic types:

1. Protease or proteolytic enzymes, which hydrolyze (break down) proteins into amino acids
2. Lipases, which break down lipids (fats) into fatty acids
3. Carbohydrases, which hydrolyze carbohydrates into simpler sugars

Enzymes for supplemental use can be sourced from animal, plant and microbial sources. Pancreatic enzymes, for example, which include all three types (protease, lipase and carbohydrase), are typically obtained from the pancreas of cows or pigs.

Proteolytic enzymes such as bromelain and papain are obtained from pineapple and papaya respectively. Enzymes can also be sourced from microbial or fungal sources. This group is the largest, as microbes and fungi can produce hundreds of different types of enzymes.

Digestive Versus Systemic Use of Enzymes

There are two primary ways of using an enzyme supplement: digestive or systemic, and the difference between them relates to timing. Taken with food, a digestive enzyme will help break down the food into smaller components.

When taken on an empty stomach, the enzymes will pass through your digestive system and enter your blood circulation, and when absorbed systemically, they serve as powerful proteases, dissolving things like fibrin and decreasing inflammation.

That said, whether you're using enzymes digestively or systemically, enzyme therapy will improve assimilation and elimination of components. In other words, the enzymes will break things down to their smallest constituent parts, making both assimilation of necessary components, and elimination of components your body does not need, easier.

As explained in a report¹⁰ by nutraceutical researcher Jon Barron, director of the Baseline Health Foundation, proteolytic enzymes taken for systemic benefit, meaning on an empty stomach, can help eliminate pathogens, allergens and rogue cells by destroying and digesting their protein-based shield. Systemically, proteolytic enzymes also have the ability to interfere with enzyme production caused by certain cancers, thereby slowing down the cancer's growth.

Systemic Use of Proteolytic Enzymes Combats Inflammation

In your gut, proteases or proteolytic enzymes, which break down dietary protein and protein-based foreign bodies, function as digestive aids. In your blood, however, they act as blood cleansers that combat inflammation and rebalance your immune system by:¹¹

How Enzymes Are Measured

Enzymes are measured¹³ in units called food chemical codex (FCC units) of some type of assay, such as hemoglobin unit tyrosine base (HUT). The FCC unit is essentially a measure of the enzyme's functionality — how well it functions under a specific assay or test.

The example given in the lecture is protease having a measure of 50,000 HUT, which means the protease can break down 50,000 bonds of red blood cells under certain laboratory conditions.

This is important to look for when shopping for an enzyme, as the FCC units are a guarantee of a certain level of activity. Simply knowing the weight or mass of an enzyme doesn't tell you anything about its functionality, as its activity could theoretically be zero.

Factors That Affect Enzyme Activity

As noted in the video presentation, environmental factors such as the pH level and temperature inside your body can affect the activity of enzymes. As your temperature rises, enzyme activity will typically increase. If the temperature gets too high, however, the enzyme will break down.

The reason for this is because the positive and negative charges of the amino acid bonds that give the enzyme its shape cause it to vibrate. As the temperature increases, this vibration speeds up, making the enzyme work harder. This is essentially what happens when you have a fever. As your temperature rises, your enzymes start going into overdrive to heal your body.

At a certain vibrational rate, however, it's simply vibrating too quickly to remain stable, causing it to break apart. This is not a concern for most supplements though, as most remain viable up to 120 degrees Fahrenheit, and the inside of your body will never get that high. Improper storage, on the other hand, could inactivate the enzymes in the supplement if it gets too hot.

As you'd expect, with lower temperature, enzymatic activity decreases. Many will store their enzyme supplement in the refrigerator or freezer for this reason, but this actually isn't a good idea. The reason? Because taking the bottle in and out of the fridge or freezer could introduce moisture, and this moisture (water) will activate the enzymes.

The best place to store your enzyme supplement is in a relatively cool, dry area such as a kitchen cabinet or pantry. Properly stored, an enzyme supplement will typically retain full potency for up to a year.

The other factor that affects enzyme activity is your body's pH (acidity versus alkalinity), which changes throughout your digestive tract. As a result, a particular enzyme will be most active or effective in a particular part of your digestive tract, and less active in others.

For this reason, high-quality supplements will contain enzymes with a wide range of pH tolerance, thereby allowing the supplement to perform optimally all the way through your gastrointestinal tract.

Why Use Enzyme Supplementation?

As mentioned, your body naturally produces enzymes. So, why would you ever need an enzyme supplement? While it's true your body continually produces enzymes, certain factors can limit this capacity:

While aging is inevitable and genetics cannot be altered, you still have a great degree of influence over your enzymes via your lifestyle choices. The healthier your lifestyle, the better your enzymatic activity will be, even without assistance from a supplement.

For example, eating plenty of fresh, raw and/or fermented foods will supply your body with healthy enzymes. Sprouts are a particularly excellent source of live enzymes.

Fasting has also been shown to conserve enzymes. If you do not eat, you will not produce digestive enzymes, allowing metabolic enzyme production and activity to proliferate instead. A supplement can still be valuable, however, to counteract genetics, aging and a less than ideal lifestyle.

How Digestive Enzymes Impact Digestion

When you swallow a food, it first enters the upper portion of your stomach. Here, any enzymes inherent in the food itself start to activate, helping to break the food down. As you might expect, the more the food can be broken down here in the first stage of your gastrointestinal tract, the less labor intensive the digestive process will be later on.

The pH in this upper stomach portion typically ranges from 4 to 6, i.e., slightly acidic. As food enters your stomach, proton pumps lining the lower pyloric part of your stomach starts pumping in hydrochloric acid, and it does this in proportion to the amount of food that you eat. The more food you put in, the more hydrochloric acid is being pumped in to help break down and liquefy that food.

Importantly, hydrochloric acid does not actually help you digest your food. Rather, it activates an enzyme called pepsin, a proteolytic enzyme that helps digest protein. In this lower section of your stomach, the pH ranges from 2 to 4.

As the food is liquefied, it starts dripping into the duodenum, the upper part of your small intestine, triggering your pancreas to secrete alkaline bicarbonates, thereby neutralizing the acidity. The pH of your small intestine typically ranges from 8 to 9.

Pancreatic enzymes are also released, which continue the process of breaking the food down into even smaller constituent parts. In summary, digestion can occur in three areas — your upper stomach, lower stomach and small intestine — and your food choices can significantly influence where and how well digestion occurs in these areas.

Enzyme supplements can also influence digestion in each of these areas, and help optimize assimilation and elimination of the foods you eat. As mentioned, for optimal benefit, you want a supplement capable of working in a wide range of pH levels, as your stomach and small intestine ranges from 2 on the acidic side to about 9 on the alkaline side.

More often than not, taking a blend of enzymes is also beneficial, as no single enzyme can perform all the necessary functions throughout your digestive tract. As noted in the featured lecture, "the better the blend, the better the breakdown of the substrate" into single, di- and tri-chain amino acids.

Summary of What to Look for in an Enzyme Supplement

In summary, factors you want to look for when buying an enzyme supplement are:

• Blends of enzymes rather than single enzymes
• Enzymes that work across a wide range of pH levels
• FCC measurements rather than just weight, as this guarantees potency (higher FCC units indicating higher enzyme activity)

Contraindications for Proteolytic Enzymes

While proteolytic enzymes are well-tolerated and safe for long-term use in most people, there are exceptions. If any of the following scenarios apply to you, you should not take proteolytic enzymes:¹⁴

You're on prescription blood thinners such as Coumadin, Heparin or Plavix

You're having surgery within two weeks (as they can increase surgical bleeding)

You have a stomach ulcer

You're pregnant or lactating

You're currently taking antibiotics

You've had an allergic reaction to pineapple or papaya

If you're currently taking a nonsteroidal anti-inflammatory drug for pain and want to add a systemic enzyme, be sure to take them at least one hour apart from each other.

Systemic enzymes are in many ways preferable to painkillers since they effectively lower inflammation and support your body's innate ability to heal itself, while pain medication simply masks the symptoms while raising your risk for addiction and death.

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Judy Mikovits, Ph.D. is a cellular and molecular biologist,¹ researcher and founding research director of the Whittemore Peterson Institute that researches and treats chronic fatigue syndrome (CFS) in Reno, Nevada.

She is likely one of the most qualified scientists in the world to comment on this disease because of her groundbreaking research in molecular biology and virology. Mikovits is absolutely brilliant but, like many gifted researchers, her complex discussions on science are quite challenging for the average lay person to follow.

For this reason, I present her interview in a different format, cutting and splicing pieces together to present a more cohesive and coherent presentation of her many important points. I would encourage you to watch the initial, very short, videos first, so you will be well-grounded, and if you are motivated, watch the entire interview at the bottom of this article.

Because there were so many surprising and important revelations in this interview I will present part 2 next week along with an interview with Bobby Kennedy Jr. which will revolve more on the vaccine issue.

Mikovits Doesn't Believe SARS-CoV-2 Is the Cause of COVID-19

One of the most shocking revelations Mikovits reveals is that she doesn't believe SARS-CoV-2 is the cause of COVID-19 but merely serves to activate or wake up a dormant XMRV infection. To support her assertion, she states that COVID-19 patients have the same cytokine signature as the gammaretrovirus XMRV, which she published many years ago.

XMRV stands for "xenotropic murine leukemia virus-related virus." Xenotropic refers to viruses that only replicate in cells other than those of the host species. So, XMRVs are viruses that infect human cells yet are not human viruses.²

The XMRV retrovirus is actually the virus that has the same cytokine storm signature as COVID-19, not coronaviruses, which are far more benign. (I delve into what retroviruses are in another section further below.)

Additionally, there may be other infections that also are contributing to the infection, such as Borrelia and Babesia or parasites, which may be why some of the antiparasite drugs like Ivermectin and hydroxychloroquine are working.

Vaccine Gammaretroviruses Have Adapted and Are Aerosolized

Getting back to the issue of gammaretroviruses, Mikovits' research showed that many of our vaccines are contaminated with them. How did this happen? In short, vaccine viruses were replicated and grown in animal cell cultures that were already contaminated with retroviruses. In other words, the root of the problem stems from the use of contaminated cell culture lines.

Vaccine manufacturing frequently involves the use of animal tissues and many vaccines are grown in animal culture cell lines. As noted in the 2010 paper, "Of Mice and Men: On the Origin of XMRV," published in Frontiers in Microbiology (which Mikovits did not work on):³

"The novel human retrovirus xenotropic murine leukemia virus-related virus (XMRV) is arguably the most controversial virus of this moment. After its original discovery in prostate cancer tissue from North American patients, it was subsequently detected in individuals with chronic fatigue syndrome from the same continent …

The detection of integrated XMRV proviruses in prostate cancer tissue proves it to be a genuine virus that replicates in human cells, leaving the question: how did XMRV enter the human population?

We will discuss two possible routes: either via direct virus transmission from mouse to human … or via the use of mouse-related products by humans, including vaccines. We hypothesize that mouse cells or human cell lines used for vaccine production could have been contaminated with a replicating variant of the XMRV precursors encoded by the mouse genome."

Mikovits goes even further, explaining that, "It became clear in 2011 that these [gammaretro]viruses had adapted to become aerosolized." This is a rather shocking finding, and this, Mikovits says, is what allows the gammaretroviruses to spread in laboratories from one cell line to another.

This could be related to research catalyzed by Charles Lieber, the former head of Harvard's chemistry department, who is a nanoscience expert and was arrested by federal authorities in January 2020 for working with the Wuhan Virology Institute.

Lab workers may also be inadvertently spreading them as they are using cell lines contaminated with retroviruses in vaccine production that could result in the spread of these retroviruses via the finished vaccine. Mikovits suspects COVID-19 may in fact be a type of vaccine-derived or vaccine-induced retroviral infection.

"I don't believe [COVID-19] is infection from without," she says. "I believe the spread across [210] countries⁴ is from injection, and there's enough evidence to support that."

SARS-CoV-2 — A Combination of SARS, Gammaretroviruses and HIV

Another of her theories is that SARS-CoV-2 is unlikely to have had a zoonotic origin but is likely synthetically produced. She believes it originated in and escaped or leaked from a biosafety laboratory. Mikovits believes both scenarios might be at play, where a lab-created virus, SARS-CoV-2, is causing serious infection and/or death only in those who have underlying retroviruses in their bodies.

Mikovits suspects that people who do not have retroviral infections, SARS-CoV-2 causes no or only mild symptoms. Another possibility is that the SARS-CoV-2 virus is the result of growing coronaviruses in retrovirus-contaminated cell lines, producing a gammaretrovirus-carrying virus.

According to Mikovits, her 2009 through 2011 work suggested 25 million to 30 million Americans were carriers of XMRVs and other gammaretroviruses. That estimate is over a decade old now so the number is likely far higher.

"There is a family of gammaretroviruses, most likely [in] contaminated blood supply and vaccines that are still to this day, almost 10 years later, being injected," she says.

"We don't need an infectious virus if you inject the blueprint, if you inject the provirus. And … there are a lot of data to support COVID-19 is not SARS-CoV-2 alone, that it's SARS-CoV-2 and XMRVs (human gammaretroviruses) and HIV."

Might Wearing a Mask Worsen Your Odds of Illness?

Mikovits is also highly critical of the recommendation (and in some places mandate) to wear a face mask or fabric cover such as a bandana around your face. She believes:

"Wearing a mask is going to cause more secretions and give more cells a home and amplify any viruses. [Wearing a mask is] immune suppressive; it's going to limit your body's ability to produce Type 1 interferon.

You're driving the infection in yourself and you're not preventing the spread. [Instead], you're amplifying [replication of] not just [SARS-CoV-2] but also many other [viruses], including your XMRVs, influenza or other dormant viruses.

What keeps those dormant viruses dormant? Your natural killer (NK) cells, your mast cells, your macrophages. That's where you're getting the inflammatory signature.

So, every virus you amplify is driving the inflammatory signature, and you're going to get sick. [The resulting illness] doesn't have to be SARS-CoV-2 at all. You're making yourself sick [by bringing dormant viruses out of dormancy]. It's insanity."

Wearing a face mask after getting a live flu vaccine may further worsen your odds, she says. Why? Because you're injecting three or more live flu virus strains into your body, which lowers your immune function. You're also going to shed the viruses contained in the vaccine. If you wear a mask, Mikovits says, you'll shed those viruses into the mask, which could encourage illness.

On the other hand, not wearing one might jeopardize the health of others. "If you're shedding [the viruses] into the air, you're going to make somebody else get another upper respiratory infection that's going to allow [SARS-CoV-2] to make them sicker," she warns.

Why PCR Testing Is a Bad Idea

We're also being lied to about the prevalence of infection. At the height of the pandemic, we saw inflated case numbers for the simple reason that the Centers for Disease Control and Prevention stopped requiring doctors to do testing in order to confirm that a patient is in fact infected with SARS-CoV-2 or died from COVID-19. The numbers now include "suspected" and "assumed" cases.

Naturally, without widespread and accurate testing, there's no way to get a clear idea of how prevalent the infection is, and how many actually get sick and die from it. The initial emphasis on PCR testing resulted in massive false positives and greatly inflated numbers of those infected.

As noted by Mikovits, confirming each case through testing matters greatly, as there are hundreds, if not thousands, of microbes that can cause upper respiratory infections, including seasonal influenza viruses. None of those should be lumped in with COVID-19 if we want to understand the true nature and danger of this disease.

What's more, the initial decision to use RT-PCR (reverse transcription polymerase chain reaction) testing instead of antibody testing was an unwise one, as it virtually guaranteed an overestimation of the problem. RT-PCR is now being used to diagnose an active infection by detecting the presence of SARS-CoV-2 genetic material.⁵ However, by doing that, you end up with high rates of false positives. Mikovits explains how the RT-PCR test works:

"We're taking a swab and scraping some epithelial cells [from the back of the sinuses or throat] because that's what coronaviruses infect … We get a little RNA — because it's an RNA virus — we reverse-transcribe that, meaning write it backwards with enzymes in the lab, and then we amplify it [through a] polymerase chain reaction …

We're only taking a piece of the virus, we're not taking the whole virus … The first thing about [the PCR] test is, it was admitted by the U.S. Food and Drug Administration and the CDC that the tests put out by the CDC were contaminated.

And when you amplify something a million times, or 10 million times — whatever they do in the 30 cycles or so — it's logarithmic that RNA then is way overestimated … [But] no [viral] particle was identified or isolated from your saliva or from your nasal passages. Nobody took the secretions from your nose or your mouth and isolated the [actual] viruses.

[When I isolated] HIV in 1983, I isolated it from saliva. What you do is you take the virus and grow it in any human cell, in an appropriate cell line, and you make many copies. [Viral replication] means you have [a positive test for] that virus. Then you sequence the whole virus.

A PCR [test, on the other hand] can give you a lot of false positives [by amplifying RNA fragments].

We [also] showed the people that had [HIV] infection had antibodies; that they had been fully exposed and it was not a piece of nucleic acid in a biopsy or in their throat or in their nose. [A piece of nucleic acid] is not a virus. And it's certainly not infectious.

If RNA is there and in the tiniest amount, I'm not going to cough it on somebody, especially if I'm not coughing. I'm not going to breathe that [out and infect] somebody because there's no evidence of an infectious virus."

Better Testing Strategy: Antibodies

Rather than using PCR testing, "what should have been done is test for antibodies," Mikovits says. This is what was done in South Korea. An antibody test will tell you whether you had the infection at some point, and have developed a strong immune response or immunological memory that will allow you to fight the infection should you encounter it again.

"Epidemiology is not done with PCR. In fact, Kary Mullis who invented PCR, Nobel Laureate, and others, said PCR was never intended for diagnostic testing. So that puts that to bed.

It takes nothing to develop a really good serology [i.e., antibody] test … [It takes] a few weeks. It's pretty easy because the people who have recovered have antibodies. So, you isolate those antibodies, you take their plasma, you purify the antibodies, and then you can grow them.

Then you develop the tests... It's usually ELISA or Western Blot [which check for] the protein and the antibody binds. You form an immune complex, and you detect it with a dye. You can do that test with a finger stick … and it takes 15 minutes to get the answer, almost like a pregnancy test."

My belief is that the use of PCR instead of a proper antibody test was intentional, as it inflates the case numbers. Mikovits agrees, saying "I wouldn't get any tests right now. I'd simply wash my hands and drink hot lemon water as I always do for any flu season."

Evidence SARS-CoV-2 May Be a Lab-Created Virus

In the Epoch Times documentary, "Tracking Down the Origin of the Wuhan Coronavirus,"⁶ Mikovits details some of the evidence supporting the view that SARS-CoV-2 is not a naturally-evolved virus, but rather a laboratory concoction.

One piece of evidence is that the virus contains a protein envelope from the HIV virus. It's also very similar to SARS which, according to bioweapons expert Francis Boyle, is an engineered bioweapon.

As explained by Mikovits, an Indian paper^7,8 detailed the presence of Gp120, a protein envelope from the HIV virus. That paper was quickly retracted due to political pressure. However, Mikovits colleague, Luc Montagnier, made a similar discovery, finding Gp41 in the SARS-CoV-2 virus, which is the transmembrane domain of the HIV virus.

"The folks from India also had GAG. That's structural proteins. That gives you a clue that it wasn't a CRISPR technique or a pseudotyping where the envelope was expressed in a gene therapy-type of way. If it were CRISPR, you wouldn't put the GAG sequences in there.

What was done is, the virus was acquired as they grew SARS-CoV-2 in Vero-E6 cells — the monkey kidney cells where you get HIV.

Simian immune deficiency virus was the origin, and we were told all the way back in the 80s that somebody forgot to cook their food in Africa and a few promiscuous men spread this [HIV] virus around the world. So, you can see again the patterns of the lies and of what people end up believing."

The addition of this envelope protein from HIV gives SARS-CoV-2 the ability to impair the immune system. It also contributes to its pathogenicity. Mikovits continues her explanation:

"The first thing is, you must grow a virus to make a lot of it. So, you grow it in cell lines. They didn't take [SARS-CoV-2] from the bat and it jumped into a human. It normally goes through another cell [from] a monkey or a smaller animal. The cell line that supports the growth and expansion [of viruses] are monkey kidney cells.

Maybe [SARS-CoV-2] is not engineered at all … but the end result is, now it not only infects the epithelial cells of the lungs, it infects the white blood cells, it infects the immune cells. We see the splenomegaly in large spleens, we're seeing penias, cytopenias. We're losing cells like HIV-killing T-cells …

So, it's got not only an expanded host range, but also disease symptoms that make no sense for a coronavirus.

Hence, we're killing people because they're treating an upper respiratory infection, and you're getting that inflammatory disease signature because you're infecting the very innate immune response, the macrophages, the monocytes, the natural killer cells, the T cells. And it's primarily the T-cells in the macrophages because those are the cells HIV 120 and Gp41 infect through CCR5 in the CD4 receptor.

So now you're going to lose your adaptive immune response, you're going to drive the inflammation. And it's the fire [of inflammation] that does the tissue damage."

Another piece that hints at SARS-CoV-2 being a manufactured virus is the construction of its spike proteins, which bind to ACE2 receptors to gain access into the cell. This appears to be an engineering feature. According to Mikovits, it's quite clear that the spike proteins came from the original SARS virus, which also infects through ACE receptors.

There are also "single point mutations there that make it far more infectious, easier to spread," she says, "and how those were acquired, nobody really can say." At least not yet. Nanotechnology may also have been used to aerosolize it for ease of transmission.

"The nano[size] further increases the host range. So now you can go into every cell. Now you can go across the blood brain barrier. That's nano. Now you don't need a receptor. You can breathe it, it can go into every cell of the body. You don't need the gatekeeper. You don't need the receptor. You don't need the lock and key."

Contaminated Cell Line Shared With Wuhan Biolab

According to Mikovits, one contaminated cell line is the Vero monkey kidney cell line called Vero E6, which was given by Fort Detrick — a U.S. Army Medical Command installation that hosts many of our national biological defense programs and houses the National Cancer Institute laboratory where she used to work — to the biosafety 4 laboratory (BSL-4) in Wuhan, China. This cell line is what the Wuhan lab used to grow and study coronaviruses, she says.

The Vero cell line is listed in the 2015 paper,⁹ "A SARS-like Cluster of Circulating Bat Coronaviruses Shows Potential for Human Emergence," co-written by University of North Carolina researchers and Dr. Shi Zhengli, a Chinese virologist at the Wuhan lab who in 2010 published a paper¹⁰ discussing the weaponization of the SARS virus.

The contaminated Vero monkey kidney cells were also used in the production of polio vaccines, Mikovits notes. The original polio vaccines were passed through mice brains, as we didn't have cell lines in the 1930s when that vaccine was originally developed. According to Mikovits, the spread of this Vero retrovirus has occurred through laboratory workers and hospital caretakers for decades.

"That's why the family studies we did were so important," she says, referring to studies in which retroviral transmission was tracked to determine how it spread between family members.¹¹

Alas, whenever patterns were detected, she was always directed to cover them up. Her refusal to hide the information from the public was what led to her firing in 2011. According to Mikovits, we're seeing the same pattern of sweeping evidence under the rug now during the COVID-19 pandemic.

"The patterns are the same as far as the science goes, and the patterns are the same as far as the political corruption, the plague of corruption, in covering up data," she says.

Mikovits Pioneering Research in XMRV

In 2009, Mikovits got embroiled in controversy when she wrote a paper reporting that a retrovirus known as xenotropic murine leukemia virus-related virus may play a causal role in CFS and other diseases, including autism.

Her career background and past troubles also involved Fauci who, according to Mikovits, is guilty of scientific fraud. She details this in her book, "Plague of Corruption: Restoring Faith in the Promise of Science."

According to Mikovits, Fauci does not appear to have changed his stripes, and is still misleading the public and hiding the truth about SARS-CoV-2, just like he did with the HIV virus and retroviral-associated diseases.

"I think the way to think about the background of what's going on right now is to go back to my first interactions with Dr. Tony Fauci when I was a 25-year-old lab technician in the National Cancer Institute. At that time, we had isolated — from blood and saliva — the lymphadenopathy virus.

[Lymphadenopathy-associated virus (LAV)] was the name given to it by Luc Montagnier, the Nobel Laureate, [who] first isolated and discovered that virus and its association with HIV/AIDS.¹²

In that situation, Fauci delayed the serology testing [to find out] who was exposed [to HIV]. It was politicized such that the only people that were [said to be] susceptible to getting infected with HIV was gay men [and] IV drug users.

The country was told not to worry about it. It was only spread through blood and body fluids and shouldn't be a problem for most other people. So, the testing that could have been done wasn't done because of political reasons, and the treatments weren't done because Fauci had patents, and — we didn't know this at the time — the wrong type of treatment was used. That led to the spread and [death] of millions worldwide …"

The Discovery of Human Gammaretroviruses

Ultimately, Mikovits and her colleagues discovered that the HIV virus was spread through a contaminated blood supply. After that, they proceeded to look into other "clearly retroviral-associated diseases," such as CFS,¹³ certain kinds of autism, cancers, leukemias and lymphomas.

Gammaretroviruses¹⁴ are viruses that can cause cancer, leukemia and immune deficiencies in various animals. Examples include murine leukemia virus, feline leukemia virus and mink focus forming virus. As explained in a 2011 paper on gamma retroviruses:¹⁵

"Retroviruses are evolutionary optimized gene carriers that have naturally adapted to their hosts to efficiently deliver their nucleic acids into the target cell chromatin, thereby overcoming natural cellular barriers …

Retroviral vectors are fascinating and efficient delivery tools for the transfer of nucleic acids. As a hallmark, all retroviruses are capable of reverse transcribing their single stranded RNA genome into double stranded DNA, which will be stably integrated into the host cell genome.

As highly evolved parasites they act in concert with cellular host factors to deliver their nucleic acid into the nucleus, where they exploit the host cell's machinery for their own replication and long-term expression occurs."

The key take-home here is that retroviruses are "integrated into the host cell genome," and infection can result in "long-term expression." In other words, once they're in your body, they can remain dormant, only to reactivate when conditions are favorable. In this regard, they're quite different from your average virus that, when you're exposed, invades your cells, replicates and causes symptoms, and is eventually eliminated from your body through your immune response.

In 2009, Mikovits and her team discovered and isolated the first human gammaretrovirus family of retroviruses, known then as XMRVs. As mentioned earlier, XMRV stands for "xenotropic murine leukemia virus-related virus." Xenotropic refers to viruses that only replicate in cells other than those of the host species. So, XMRVs are viruses that infect human cells yet are not human viruses.¹⁶

My Entire Interview With Judy Mikovits

To reiterate some of the key take-home messages Mikovits delivers in this interview:

Last but not least, if this topic intrigues you, be sure to pick up a copy of her new book, "Plague of Corruption: Restoring Faith in the Promise of Science." You can also find more information on her website, plaguethebook.com.

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When Emma Carey’s parachute failed to open during a skydive over the Swiss Alps, the young Australian was convinced she was going to die. Against all odds, she survived. Then, her doctors told her she would never walk again. This time, however, she was convinced she was going to thrive. In this exclusive interview with Body+Soul, Carey shares her journey so far and how she’s learnt to stand on her own two feet – in more ways than one.

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New findings have demonstrated a molecular link between COVID-19 and serotonin cells in the gut. The research could help provide further clues to what could be driving COVID-19 infection and disease severity and supports previous evidence that antidepressants, known as selective serotonin reuptake inhibitors (SSRIs), could reduce the severity of COVID symptoms.

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