Health & Fitness

This article was previously published July 19, 2020, and has been updated with new information.

Tyler W. LeBaron, founder of the science-based nonprofit Molecular Hydrogen Institute, is one of the most knowledgeable people about molecular hydrogen and its benefits. There are so many benefits we can learn from him and many other researchers from Universities around the world. For starters, H₂ is a potent selective antioxidant. This is important, as many other antioxidants, such as vitamin C and E are not selective, and when taken in excess, can be counterproductive.

Hydrogen doesn't have that downside, which is one of the reasons why it's one of my favorites. Now, when we talk about molecular hydrogen, we are talking about the gas, the H₂ molecule, which is two hydrogen atoms bound together.

The H₂ molecule is the smallest in the universe, which allows it to diffuse through all cell membranes, including the blood-brain barrier and subcellular compartments, and into the mitochondria. It doesn't need any transporter protein.

It also has no charge or polarity. As explained by LeBaron, that's critical, because charged molecules cannot easily penetrate cell membranes. Charged molecules must go through a protein channel. All of this gives it superior cellular bioavailability.

Health Benefits of H₂

Among the many health benefits of H₂ is its ability to decrease excessive oxidative stress, inflammation and perturbations from normal homeostasis. The key word here is "excess," because some oxidative stress and some free radicals are actually beneficial. For example, you metabolize food through the process of oxidation, and that oxidation is necessary for life to exist.

So, what we're looking for in terms of health is the ability to inhibit excessive oxidative stress and damage. LeBaron reviews this in greater detail in the interview so, for more information, please listen to it in its entirety, or read through the transcript.

As just one example, certain therapies such as photobiomodulation, exercise and sauna bathing mildly increase oxidation in the body, and that oxidation is what induces various beneficial effects such as the induction of heat shock proteins (HSP). This process is known as hormesis.

"This is an important word … when we talk about the benefits of molecular hydrogen because it seems to work through some similar processes of hormesis," LeBaron says.

H₂ Is a Selective Antioxidant

When it comes to oxidative stress, all you really want is a return to homeostasis. You don't want to neutralize all free radicals. Many antioxidants have a high number of electrons that can easily and indiscriminately scavenge, react with and neutralize a wide range of radicals or oxidants. Molecular hydrogen, on the other hand, is selective, and thus only eliminates the excess, so that homeostasis is restored.

"Sometimes antioxidants can even exacerbate oxidative stress because they can increase Fenton reaction cycles and redox cycling, and end up being potent pro-oxidants. So, it is very complicated, and we have to be very cautious," LeBaron says, adding:

"One of the reasons we know H₂ [is] safe is because it simply does not have the reductive power or potential to neutralize or react with some of these critical important signaling oxidants, such as hydrogen peroxide, superoxide radicals and nitric oxide. It just does not have the ability to react with these, even in vitro. If you just put the two together, they don't react."

On the other hand, H₂ readily reacts with the toxic hydroxyl radical — the most reactive and oxidative radical in the body — turning it into harmless water. Studies suggest H₂ may be very helpful in cases of heart attack or stroke, for example, protecting against the oxidative damage from hydroxyl radicals that occur during reperfusion.¹ In my view, molecular hydrogen should be implemented ASAP in all cases of heart attack and stroke for this reason.

There's no risk, it's very inexpensive and the upside potential is enormous. LeBaron cites animal research published in the Journal of the American Heart Association² showing H₂ administration increased the post-cardiac arrest syndrome survival rate from 43% in the control group to 92% in the H₂ group. When combined with therapeutic hypothermia, which inhibits the creation of free radicals, the survival rate shot up to 100%. It simply doesn't get any better than that.

According to LeBaron, the Japanese government has now approved the inhalation of H₂ gas as an advanced medicine for the treatment of post-cardiac arrest syndrome.³ He also reviews some of the studies that are currently underway to investigate the benefits of molecular hydrogen inhalation during heart surgery and other instances.

H₂ Is a Signal Modulator

Aside from being a selective antioxidant, H₂ acts as a gaseous-signal modulator, and thus is able to influence gene expression and protein phosphorylations cascades involved in signal transduction, all of which help explain its therapeutic effects. One of the primary pathways that H₂ activates is the Nrf2 pathway. LeBaron explains:

"The Nrf2 is this protein that's bound to another protein, Keap1, and when there's an assault of oxidative stress, those two separate. Then the Nrf2 is able to diffuse into the nucleus of the DNA. It binds to the electrophile response or ARE, the antioxidant response element, portion of the DNA.

When it does that, that ends up leading to the production of a whole bunch of endogenous antioxidants like glutathione, superoxide dismutase and catalase … When we talk about antioxidation and detoxification, a lot of that is regulated and controlled by Nrf2. That is the master regulator. So, it is a key protein involved in many processes [and] hydrogen gas is able to activate the Nrf2 pathway."

Importantly, though, contrary to other Nrf2 activators, H₂ only activates Nfr2 if it's actually needed. In this way, the risk of it suppressing beneficial free radicals like nitric oxide is minimized. Indeed, H₂ appears to be one of the safest therapeutic options available. It's downside potential is almost nonexistent.

"It tends to bring things back to homeostasis," LeBaron says. "The further something is away from homeostasis, the higher the probability that hydrogen gas will be able to help bring that back into homeostasis. If something is already at a perfect level, well, then, you may see that hydrogen gas didn't do anything …

Again, hydrogen gas has this dual role where it can both protect against the oxidative stress, as well as act as this mild hormetic effector in the mitochondria to increase mild amounts of free radicals, similar to an easy bout of exercise for example, which can then induce these protective effects."

How to Administer H₂

The easiest way to get hydrogen gas into your system is to dissolve a molecular hydrogen tablet in water and drink it. In the interview, LeBaron warns us why we need to be skeptical and cautious about electrolysis machines, as they often don't produce anywhere near the concentrations required. In clinical studies this is often 1.6 mg/L and above, which at first doesn't sound like very much, but it is significant as LeBaron further explains:

"There are a couple of things to consider when you drink hydrogen gas. No. 1, 1.6 mg/L as a solubility doesn't sound like very much … [but remember] that hydrogen gas is the smallest molecule in the universe. Of course, 1.6 mg doesn't weigh very much because it's hydrogen gas … but it's actually a lot of molecules. In fact, there are more molecules in 1.6 mg of hydrogen than there are molecules of vitamin C in a 100-mg dose.

You have to compare molecules to molecules or moles to moles, not just weight to weight. What weighs more, a pound of gold or a pound of feathers? Right? They weigh the same … So, when we look at molecular hydrogen, there is actually quite a bit.

Now, get this. When you inhale, say, a 3% hydrogen gas, then that's going to increase the cellular concentration to a certain level. That exact same level, if we can calculate it based on Henry's law and the dose you're ingesting from drinking hydrogen water, that concentration in the cell can also be reached by just drinking hydrogen water.

Because if you drink all of it at once … [it] immediately increases the cellular concentration to the same level that you would get if you were inhaling hydrogen gas at 2% or 3% level … You're also able enact various second messenger systems that maybe you're not getting with inhalation."

Research has shown H₂ water can improve nonalcoholic fatty liver disease⁴ and metabolic syndrome,⁵ both of which are diet-driven conditions. In a recent study⁶ looking at metabolic syndrome, a high dose of H₂ was used using hydrogen-producing tablets.

The study involved 60 subjects and lasted for six months and "significantly reduced blood cholesterol and glucose levels, attenuated serum hemoglobin A-1c, and improved biomarkers of inflammation and redox homeostasis." It even "tended to promote a mild reduction and body mass index and hip-to-waist ratio," the study authors added.

"It appears we had some very prominent effects, and even more effective compared to the previous studies leading to this trend that at least in some cases, a higher dose or a higher concentration of hydrogen is more effective than the lower dose, lower concentrations," LeBaron says.

Concentration and Frequency Matter

Aside from making sure the concentration is sufficiently high, you also want to pulse your intake, as the more continuous the exposure, the less effective it is. LeBaron further explains:

"Let's say [you take] 6 mg of hydrogen and you're going to take all 6 mg evenly in a 24-hour period. That means you're essentially sipping on hydrogen water throughout the day.

If you do that, you may not get as good of benefits because you're not getting a high enough dose of hydrogen in the body in order to reach the cellular concentrations required to induce those changes at the cellular level that you need.

Now, if in contrast, if you were to just take the full 6 mg all at once, that is probably going to be more effective than taking it throughout the entire day. So, I will say if you are going to get hydrogen and try to get the benefits, then you would want to get as high of a dose you can all at once, and then you could probably do that multiple times a day.

I don't know if it's better to take 6 mg or 10 mg of hydrogen once a day or six times a day. Maybe the six or 10 times a day is going to be more effective, or just as effective, because you're still getting spikes. But then again maybe not."

Clearly, the studies need to be done to determine the best frequency, but until then, it would seem that customizing the dose to your personal circumstances might be more appropriate. So, if you're in normal, nonstressful circumstances at home, not exercising much at all, then maybe once day is sufficient.

On the other hand, if you exercise vigorously then it might be more appropriate to take it a couple of times a day. If you travel by airplane, taking it every two hours while flying might be appropriate. The good news is, H₂ is quite safe, so you're unlikely to do harm.

Another benefit when using hydrogen tablets is that they contain highly bioavailable unbound magnesium ions. Each tablet will provide about 80 mg of ionic magnesium, which is about 20% of the RDA.

Synergistic Effects With Other Therapies

H₂ gas can also be used together with other supplements and therapies for a potential synergistic effect. For example, you can take it along with a sauna, both of which produce heat shock proteins, or with nutritional ketosis or exogenous ketones. Another example is hyperbaric oxygen therapy.

"When it comes to the sauna, I think that's great," LeBaron says. "I probably would do the hydrogen before anything … Again, [we're] talking about this preconditioning hydrogen effect.

If I can just back up and talk about one study that I think helps at this stage, about NAD+ and NADH. These are very important molecules. The higher the ratio of the NAD+ to NADH, the better … In this interesting study,⁷ they used a toxin in a cell culture, and as would be expected, that NAD+ to NADH ratio decreased, and that ends up causing all of these pathological problems and cell death.

When they administered the hydrogen gas, it helped maintain those levels up higher. Now, this is part of the issue: For part of the study, they just did it in cell cultures, so you can imagine this little Petri dish, and you add hydrogen gas in there.

Well, that hydrogen gas will only be in there for 20 minutes, half an hour or 40 minutes, depending on the concentration. It's not going to be there for very long. They found there was a therapeutic protective effect against that toxin for about 24 hours. It maintained that effect …

Then there was a clinical study on rheumatoid arthritis⁸ where they used high-dose hydrogen water for four weeks. After four weeks, there was still a protective effect of molecular hydrogen. There were still decreases in the disease rating score and oxidative stress. So, it really had an effect on gene expression, epigenetics and signal modulation. Much more is going on here than just a radical scavenging activity.

Taking these together, when we look at other things such as the sauna, the sauna really is quite a mild thing … [but] I still like the idea of taking the hydrogen before. When you're talking about hyperbaric oxygen, then I think there's even more rationale of taking the molecular hydrogen [30 to 60 minutes] before as a pretreatment, preconditioning …

Ketones, whether they're endogenous or exogenous, are very beneficial for the mitochondria, as long as the mitochondria are ready for them. Ketones can also increase free radicals, at least initially, but this is also what's very good, because in the long run they can decrease oxidative stress. Part of this is why you can upregulate the Nrf2 pathway.

Well, hydrogen gas being able to both suppress excessive oxidative damage as well as improve and activate the function of the mitochondria, improving the mitochondrial resting membrane potential, it will have influence in the mitochondria transition pore, so you don't have pathological problems …

So, there are some areas where ketones seem to work, as does hydrogen gas … Hydrogen [can also] induce and actually enhance autophagy^9,10… By so doing, you're going to get therapeutic protective effects from the hydrogen gas. However, there are other studies showing that hydrogen gas inhibits excessive autophagy.^11,12

So, that's how cells die, right? You have necrosis, you have apoptosis, and you have autophagic cell death. When you have too much going on — and a lot of drugs or interventions can potentially cause an excessive amount of autophagy — then that's bad. Hydrogen gas … was able to prevent the excessive amount of autophagy being produced."

Similarly, H₂ gas can both increase and decrease mTOR activation,^13,14 depending on what your body needs. Ditto for IgF1.^15,16 What this means is that if you're fasting or doing time-restricted eating, which activates autophagy, taking molecular hydrogen not only can optimize autophagy, but also lower it if too much is taking place. That could make long-term fasting much safer. What's more:

"When you take hydrogen, you increase gastric ghrelin secretion.¹⁷ Ghrelin is the hunger hormone. One of the first things [that happens] when you fast is you increase ghrelin. Ghrelin is extremely neuroprotective and anti-inflammatory and has a whole bunch of benefits.¹⁸

Well, hydrogen also increases ghrelin. So, in a lot of ways, hydrogen mimics fasting from autophagy to ghrelin, to a lot of other pathways that are activated, but it depends on the condition."¹⁹

Dosing Basics

The normal dose is one tablet — which is considered an appropriately high dose — in 500 mL or 16 ounces of water. That will give you a concentration of about 10 mg of H₂ per liter (10 mg/L), which means you're getting a dose of 5 mg. As soon as the tablet has dissolved, you'll want to drink the whole glass before the cloud of H₂ gas dissipates.

The rate at which it dissolves can vary from anywhere from one to two or three minutes, depending on how cold the water is. If you put it in iced water, it's going to take even longer. Ideally, use room temperature water, as the colder it is, the longer it takes for the tablet to dissolve, and the longer it takes, the less of the gas will remain by the time the tablet is fully dissolved.

Also, use still water, not sparkling water, which has CO₂ dissolved in it, as that will disperse the H₂ gas out faster. You want to drink it quickly while it still has that milky look. The white cloudiness is the suspended hydrogen. If you wait until the water turns clear, the hydrogen gas has evaporated away. Again, if your body is under serious stress, you may take four or five tablets a day. If not, a single tablet a day would probably be sufficient.

For more information about molecular hydrogen research, visit the National Institutes of Health library²⁰ and search for molecular hydrogen. Also be sure to check out the Molecular Hydrogen Institute's website.

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While some still claim SARS-CoV-2 doesn’t actually exist, this seems to fly in the face of several well-established facts. The virus has actually been photomicrographed,^1,2 whole-genome sequences of the various strains are available,^3,4 and with the appropriate credentials anyone can obtain the live virus to conduct research.

While I am absolutely no fan of the U.S. Centers for Disease Control and Prevention, they do grow the virus in cell culture to ensure widespread availability for researchers who want to study it.⁵ Examples of research where you need the actual virus include antiviral research, vaccine development, virus stability research and pathogenesis research.⁶

What’s the Confusion?

At least part of the confusion appears to be rooted in how the term “isolated” is defined. Some insist a virus is not isolated unless it’s also purified, while others say a virus doesn’t have to be purified in order to be “isolated.”

Steve Kirsch claims to have asked several experts about this, noting that all, including Dr. Robert Malone and Dr. Li-Meng Yan, say that the virus has indeed been “isolated.” “So, it has been ‘isolated’ according to their belief in what the term means,” Kirsch writes, adding:⁷

“Others interpret the term differently and would claim the virus hasn’t been isolated. In fact, according to their definition, no virus in history has ever been isolated. That’s important to know. They use that as justification for their belief that there is no virus here since viruses don’t exist at all.”

When Kirsch asked his readers for input, one pointed out:⁸

“The real question is ... has it been isolated from a HUMAN subject w/o passing it through (say) Monkey Kidney Cells? Because there is plenty of evidence out there that says it hasn't been isolated directly (no intermediaries) from a HUMAN subject.”

According to Kirsch, the scientists he spoke with did not agree that this was a concern, and “Sabine Hazan verified that the sequence of the virus obtained from ATCC [the American Type Culture Collection, a global resource center for reference microorganisms] matched exactly what she found in people who have the virus.”⁹

As noted in Hazan’s paper, “Detection of SARS-CoV-2 From Patient Fecal Samples by Whole Genome Sequencing”:¹⁰

“Study participants underwent testing for SARS-CoV-2 from fecal samples by whole genome enrichment NGS [next-generation sequencing] (n = 14), and RT-PCR nasopharyngeal swab analysis (n = 12).

The concordance of SARS-CoV-2 detection by enrichment NGS from stools with RT-PCR nasopharyngeal analysis was 100%. Unique variants were identified in four patients, with a total of 33 different mutations among those in which SARS-CoV-2 was detected by whole genome enrichment NGS.”

Germ Theory and Terrain Theory Both Have Merit

As noted by independent journalist and political analyst Jeremy Hammond in a March 2021 interview,¹¹ the claim that SARS-CoV-2 has never been isolated and actually doesn’t exist at all is perhaps one of the most counterproductive arguments of the health freedom movement.

By insisting that there is no virus, and that COVID-19 is caused by things like 5G radiation alone, allows the mainstream media to dismiss entirely legitimate concerns about electromagnetic field exposure (EMF) and 5G — including the possibility that it might make some people more vulnerable to infections.

Like Hammond, I believe the pathogenesis of COVID-19 involves both germ theory and terrain theory, not just one or the other. “SARS-CoV-2 infection is an insufficient but necessary factor in the pathogenesis of COVID-19,” Hammond says, adding that “the virus is constantly being isolated and whole genome sequenced by scientists all over the world.”¹²

That said, environmental factors can clearly play a role, in that they can make you more or less predisposed to severe infection when you encounter this virus. This includes EMFs, toxins like glyphosate, previous vaccine injuries and much more.

Hammond argues that the “COVID-19 pandemic should be a wake-up call to the human population, and especially the populations of developed countries, about the need to focus on natural means of maintaining good health and living in greater harmony with our natural environment.”

Indeed. And, as Hammond points out, pathogenic challenge is absolutely necessary for general good health and strong immunity. When we shield ourselves too much from everyday pathogens, we make ourselves vulnerable to chronic diseases instead.

SARS-CoV-2 Genome Sequencing From Italy

As for whether SARS-CoV-2 has been isolated and exists as a viral entity, the answer appears to be yes. For example, an Italian paper¹³ published in the Journal of Virology, dated May 18, 2020, detailed the isolation and full-length genome of the virus taken from COVID-19 patients in Italy:

“At the beginning of March 2020, the first nasopharyngeal swabs positive for SARS-CoV-2 started to be detected in the Northern Eastern Region of Friuli-Venezia Giulia ... Swab contents were seeded on Vero E6 cells and monitored for cytopathic effect and by an RT-PCR protocol using primers for the N region.

Cell culture supernatants from passage 1 (P1) of four isolates were collected, and RNA was extracted with QIAamp viral RNA minikit (Qiagen) and quantified with an in vitro-transcribed RNA standard ... The quantity and quality of the RNA were assessed ... For each sample, 100 ng of total RNA was processed using Zymo-Seq RiboFree ribosomal depletion library preparation kit (Zymo Research).

All the obtained libraries passed quality check and were quantified before being pooled at equimolar concentration and sequenced ... Sequenced reads that passed the quality check (Phred score ≥30) were adaptor and quality trimmed, and the remaining reads were assembled de novo using Megahit (v.1.2.9) with default parameter settings.

Megahit generated in all cases 7 contigs with more than 1,000 bp and 100× coverage; all of these assembled contigs were compared (using BLASTn) against the entire nonredundant (nr) nucleotide and protein databases.

In all cases the longest and more covered contigs were identified as MT019532.1,¹⁴ ‘Severe acute respiratory syndrome coronavirus 2 isolate BetaCoV/Wuhan/IPBCAMS-WH-04/2019, complete genome,’ with 99% identity and 0 gaps.

The longer sequences were named hCoV-19/Italy/FVG/ICGEB_S1, _S5, _S8, and _S9 and were deposited in GISAID ... Sequence analysis showed an uneven coverage along the SARS-CoV-2 genome, with an average range from 126 to 7,576 reads and a mean coverage per sample of 1,169× ... Phylogenetic trees were inferred using the maximum likelihood method ...

The first sequences deposited in GISAID (EPI_ISL_410545 and EPI_ISL_410546) were collected in Rome from a Chinese tourist from Hubei province who got infected before visiting Italy, and another one (EPI_ISL_412974) was from a test-positive Italian citizen returning from China.

Only two sequences were reported from the Lombardy cluster (EPI_ISL_412973 and EPI_ISL_413489). In this report four additional sequences from cases epidemiologically linked to northern Italy have been examined ... Sequence analysis showed a good coverage along the SARS-CoV-2 genome for all four isolates.

Based on the marker variant S D614G, all four sequences grouped in the Bavarian rooted subclade G, which is dominant in Europe, including the sequence from Lombardy, but distinct from the three sequences mentioned above originating directly from China.

Intriguingly, the new isolates were more closely related to EPI_ISL_412973, while EPI_ISL_413489 was more distant. No evidence could be found for the putative 382-nucleotide (nt) deletion in ORF8 detected in Singapore, which has been proposed to indicate an attenuated phenotype.”

SARS-CoV-2 Genome Sequencing From Germany

Similarly, the complete genome sequence of the virus taken from a German woman has been published, this one in the journal Microbiology Resource Announcements, in June 2020.

Here, an oropharyngeal swab sample from a female patient who tested positive but had no symptoms at the time of the test was used to isolate the strain.¹⁵ Table 1 in the paper compares the nucleotide variants found in the sampled virus and those of a reference strain already logged in the gene bank.

Another paper¹⁶ in Annals of Internal Medicine, published in August 2020, isolated the virus from ocular (eye) secretions of an Italian COVID patient:¹⁷

“The patient, a 65-year-old woman, travelled from Wuhan, China, to Italy on 23 January 2020 and was admitted on 29 January 2020, 1 day after symptom onset. At admission to the high isolation unit ... she presented with nonproductive cough, sore throat, coryza, and bilateral conjunctivitis. She had no fever until day 4, when fever (38 °C), nausea, and vomiting began.

Infection with SARS-CoV-2 was confirmed by performing real-time reverse transcription polymerase chain reaction (RT-PCR) assay on sputum samples (cycle threshold value [Ct], 16.1) on the admission day, followed by viral M gene sequencing (GenBank accession number MT008022), and virus isolation on Vero E6 cell line (2019-nCoV/Italy-INMI1).

The full genome sequence was obtained from either clinical sample or culture isolate (GISAID accession numbers EPI_ISL_410545 and EPI_ISL_410546).”

Genome Sequencing From India and Colombia

SARS-CoV-2 has also been isolated from the urine of a COVID-19 patient.¹⁸ A November 2020 paper¹⁹ sought to determine “whether various clinical specimens obtained from COVID-19 patients contain the infectious virus,” and found SARS-CoV-2 RNA “in all naso/oropharyngeal swabs and saliva, urine and stool samples collected between Days 8 and 30 of the clinical course.”

Viable SARS-CoV-2 was also found in the nasal washes of ferrets that had been inoculated with urine or stool from a COVID-19 patient. The virus has also been isolated by researchers in the U.S.,²⁰ China,²¹ India,²² Canada,²³ Australia,²⁴ Korea²⁵ and Colombia.²⁶ The Colombian paper reads in part:²⁷

“Objective: To describe the isolation and characterization of an early SARS-CoV-2 isolate from the epidemic in Colombia. Materials and methods: A nasopharyngeal specimen from a COVID-19 positive patient was inoculated on different cell lines.

To confirm the presence of SARS-CoV-2 on cultures we used qRT-PCR, indirect immunofluorescence assay, transmission and scanning electron microscopy, and next-generation sequencing.

Results: We determined the isolation of SARS-CoV-2 in Vero-E6 cells by the appearance of the cytopathic effect three days post-infection and confirmed it by the positive results in the qRT-PCR and the immunofluorescence with convalescent serum.

Transmission and scanning electron microscopy images obtained from infected cells showed the presence of structures compatible with SARS-CoV-2. Finally, a complete genome sequence obtained by next-generation sequencing allowed classifying the isolate as B.1.5 lineage.

The evidence presented in this article confirms the first isolation of SARSCoV-2 in Colombia. In addition, it shows that this strain behaves in cell culture in a similar way to that reported in the literature for other isolates and that its genetic composition is consistent with the predominant variant in the world.”

If Virus Exists, Why Aren’t Certain Studies Done?

As mentioned earlier, the actual virus is needed in order to conduct certain studies. Now, since the virus does exist, we also ought to be able to conduct studies to assess whether the COVID shots cause antibody dependent enhancement (ADE).

As suggested by Kirsch,²⁸ “Give the vaccine to the animals, wait, then expose them to the virus” and see what happens. Does it prevent infection and transmission, or does it make the animals more prone to infection? If the animals got sicker, that would be evidence of ADE, a problem that has plagued coronavirus vaccine research for decades.

It’s why we don’t have a vaccine against the common cold, caused by coronaviruses. Remarkably, this animal research has never been done for the COVID shots. The question is why? Kirsch believes the answer is because “nobody wants to know the answer ... The top management of the FDA knows it would kill the vaccine program if they did this.”

On the other hand, the vaccinated, just like the unvaccinated, tend to experience only mild symptoms with Omicron. So, perhaps the shots aren’t causing ADE (which could turn even a milder variant into something deadly).

However, ADE is far from the only concern. Clearly, these shots are associated with a dramatically increased risk of cardiovascular, cardiac and neurological problems. These too could be confirmed through animal studies — rather than testing on our children — and we wouldn’t even need the virus for those.

Either way, I believe it’s scientifically accurate to claim that SARS-CoV-2 has been isolated, genetically sequenced, and that it exists as a pathogenic entity. Getting too far into the weeds of theories that refute the existence of viruses altogether will only slow down and hamper the truth movement rather than aid it along, and I would strongly discourage anyone from engaging in this highly unproductive narrative.

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