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02/05/21
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17:48 ,
A study reports that in a diverse, cross-national sample of youth, physical discipline and cognitive deprivation had distinct associations with specific domains of developmental delay. The findings are based on the Multiple Indicator Cluster Surveys, which is an ongoing, international household survey initiative coordinated and assisted by the United Nations agency, UNICEF.

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17:48 ,
Progressive vision loss, and eventually blindness, are the hallmarks of juvenile neuronal ceroid lipofuscinosis (JNCL) or CLN3-Batten disease. New research shows how the mutation associated with the disease could potentially lead to degeneration of light sensing photoreceptor cells in the retina, and subsequent vision loss.

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17:48 ,
Health care and education systems are two main pillars of a community's stability. How well and how quickly a community recovers following a natural disaster depends on the resilience of these essential social services. New research has found hospitals and schools are interdependent, suggesting their collective recovery must be considered in order to restore a community in the wake of disaster.

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A new study has found a link between high levels of air pollution at an individual's home address and an elevated risk of developing cardiovascular disease. Air pollution exposure appears to heighten the production of inflammatory cells in the bone marrow, triggering inflammation of the arteries.

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16:48 ,
Researchers brought forward the potential of high doses of a specific adjuvant molecule, namely CpG oligonucleotide, in successfully modulating the immune system's allergic response to the main cat allergen Fel d 1, thereby inducing a tolerance-promoting reaction and reverting the main hallmarks of cat allergy.

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10:48 ,
Until recently, researchers and oncologists had placed lung cancer patients into two broad categories: those who would benefit from immunotherapy, and those who likely would not. Now, researchers, using artificial intelligence (AI) to analyze simple tissue scans, say they have discovered biomarkers that could tell doctors which lung cancer patients might actually get worse from immunotherapy.

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09:48 ,
A new study improved memory of complex, realistic events similar to these by applying transcranial magnetic stimulation (TMS) to the brain network responsible for memory. The authors then had participants watch videos of realistic activities to measure how memory works during everyday tasks. The findings prove it is possible to measure and manipulate realistic types of memory.

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08:48 ,
A global team of researchers has developed a new strategy for fast and reliable antibody tests, which can quantify the immune response induced by vaccination and reveal the timeline and stage of pathogen infection. The team's one-step quantitative antibody tests are conducted using (blood) serum and are on a par with the gold-standard, enzyme-linked immunosorbent assay (ELISA) technique.

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08:48 ,
Around 260 million people, more than three percent of the global population, are chronically infected with the hepatitis B virus (HBV); in the long term, this often leads to complications such as liver cirrhosis and liver cancer. A cure is not yet possible with the available medication. Scientists have now investigated a new combination therapy that has proven highly effective in their infection model.

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07:48 ,
Gaining more fat cells is probably not what most people want, although that might be exactly what they need to fight off diabetes and other diseases. How and where the body can add fat cells has remained a mystery - but two new studies from UT Southwestern provide answers on the way this process works.

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00:22 , ,

A paper published by Yale University scientists proposes the rising number of adults and children who experience food allergies may be the result of an exaggerated activation of the body’s system that protects you against eating toxic foods.1 They write that as many as 8% of children in the U.S. have a potentially deadly response to what are classified as the major eight food allergens.

These are often referred to as the “Big 8” and include milk, eggs, wheat, soybeans, fish, crustacean shellfish, tree nuts and peanuts.2 According to data from 2010 referenced by the American Academy of Allergy, Asthma and Immunology, a study of 38,480 children under 18 years revealed the rate of food allergies fluctuated from 6% to 8.5%, depending on the age group.3

A later survey ending in 2016 of nearly identical size (38,408 children) found similar results, which identified 7.6% of children with food allergies.4 The data were gathered from parental surveys and not confirmed testing. They found the most reported allergies were peanut, milk and shellfish.

However, the researchers concluded the prevalence may be higher since they excluded approximately 4% of the reports in which the history was not consistent with a food allergy reaction. The U.S. Centers for Disease Control and Prevention reported a rising rate of food allergies in children from data gathered from 1997 to 2011.5

They found that over a 14-year period the rate of food allergies increased from 3.4% to 5.1%. According to the most recent data from 2016, that rate may have jumped again to 7.6%,6 which means the rate has more than doubled in 19 years. Yale immunobiologists propose this trajectory may be the result of an increasing amount of unnatural substances or environmental chemicals.7

Your Body Has a Food Quality Control System

The rising number of children and adults experiencing food allergies has raised concerns with physicians and researchers. In the paper from Yale University published in the journal Cell, researchers discussed the multiple sensory mechanisms your body uses to monitor what is consumed.8

These systems include smell, taste and chemosensory processes established in the gut and impacted by your gut microbiome. In this paper, the scientists argue that the body has a food quality control system in which an allergic response plays a role. The identification and response against food antigens can result in a lethal food allergy.

One prevailing theory for the rise in food allergies has been a “too clean” environment — called the hygiene hypothesis — where children and adults are no longer exposed to natural pathogens in the environment, triggering the immune system to become hypersensitive.9

Writing in Clinical & Experimental Immunology, scientists expanded the explanation to include the presence of processed foods, dishwashing detergent and other environmental chemicals, as well as a “too clean” environment with the absence of natural microbial exposure.

They argue these all play a role in disrupting the internal food quality control system, designed to help protect your body from noxious chemicals and harmful substances. The group believes this theory may lay the groundwork for future research, treatment or prevention. In a press release from Yale University, one of the authors suggested:10

“We can’t devise ways to prevent or treat food allergies until we fully understand the underlying biology. You can’t be a good car mechanic if you don’t know how a normal car works.

One factor is increased use of hygiene products and overuse of antibiotics and, secondly, a change in diet and the increased consumption of processed food with reduced exposure to naturally grown food and changed composition of the gut microbiome.

Finally, the introduction of food preservatives and environmental chemicals such as dishwashing detergents introduced novel elements for immune systems to monitor.”

When your body detects toxins have been consumed, it also activates the parasympathetic nervous system, intended to help neutralize the health threat.11 This response can trigger food allergies and a lack of natural threats can make the system hypersensitive.

The team believes the collective changes to the food supply and environment have effectively made the immune system respond to food proteins in the same way they would to protect against toxins.

More Potential Reasons for Rising Number of Food Allergies

The furor generated by soaring prices for an EpiPen, a life-saving treatment for the potentially lethal anaphylactic allergic reaction, has made it evident there is a lot of money at stake in the medical treatment of allergies.

As veterinarian Dr. Karen Becker recently covered in one of her Healthy Pets articles, this is another way pets are good for your health. Exposure to dogs and cats can influence the developing immune system of children. Two studies were presented at the American College of Allergy, Asthma & Immunology in 2017 demonstrating children born in homes with a dog had a lower risk of allergic eczema and asthma,12 supporting earlier research.13

The exposure to diverse bacteria and other microbes from dogs has led to them being called “the new probiotic.”14 Dieter Steklis, a professor of psychology and anthropology at the University of Arizona, has studied the physical and microbiome relationship in humans and pets. He spoke with a journalist from the Tucson Sentinel, saying:15

“It’s always surprised me how many diseases and disorders are linked to inflammatory processes that link back to your immune system. If having a dog actually tames your immune system, which is what it seems to do, then elderly who have a dog may have a lower chance of depressive illnesses.”

Other theories that have been proposed for rising food allergies include overuse of medications used to reduce stomach acid, as these can alter your gastrointestinal microbiome.16

Research from King's College London also proposed that when parents avoid introducing young children to foods known to produce an allergy there was an increased potential the child would have a reaction later.17 The study selected children who had a known allergic reaction to eggs or had eczema to evaluate if avoiding an allergen would increase or decrease a food allergy.

The data showed when parents avoided feeding their children peanuts, 13.7% developed an allergy by the time they were 5 years as compared to 1.9% who were introduced to peanuts earlier. Researchers are also questioning if food allergy rates are rising because we spend more time indoors, thus contributing to the fact that vitamin D levels in nearly 40% (39.92%) of the general population are suboptimal.18

Research analyzing EpiPen use in the U.S. found a strong north-south gradient, where more were used in the northern states, suggesting there were “important etiological clues (vitamin D status)” that merit further investigation.19

Similar data were found in Australia two years later when it was revealed EpiPen use and anaphylaxis admissions were more common in areas receiving less sunshine, providing “additional support for a possible role of vitamin D in the pathogenesis of anaphylaxis.”20

Is It a Food Allergy or an Intolerance?

There is a difference between a food sensitivity or reaction and a food allergy. A true food allergy is mediated by the immune system and triggered by a reaction to proteins found in a specific food or drink. Food sensitivities, also called food intolerance, is usually an unpleasant gastrointestinal reaction to something you've consumed but is not mediated by your immune system.21

For example, a true milk allergy is different from lactose intolerance.22 The first triggers an immune response and the second triggers gastrointestinal symptoms from an inability to digest milk proteins. Type 1 food allergies involve immunoglobulin-E (IgE), which is an antibody found in the blood and mast cells in all body tissues.

After you've eaten the food the first time, cells produce IgE for the food protein that triggered the reaction, called an allergen. IgE is released and attaches to the surface of mast cells. This sets the stage for the next time you eat the food that contains the specific allergen. The protein interacts with the IgE on the mast cells and triggers a release of histamine.

IgE reactions sometimes start with itchiness in your mouth, followed by vomiting, diarrhea and stomach pain. Some protein allergens can cross immediately into your bloodstream and trigger a bodywide reaction including dizziness or feeling faint, repetitive coughing, tight, hoarse throat and a weak pulse.23 It can also activate an anaphylactic reaction resulting in a drop in blood pressure, hives and wheezing.

A Type 1 food allergy can take anywhere from a few minutes to a couple of hours to develop. A second type of food allergy, Type 3, is mediated by immunoglobulin-G (IgG). This is a delayed food allergy that happens four to 28 hours after exposure.24

Adult-Onset Food Allergies Are Not Uncommon

Although most food allergies develop in childhood, it is not unheard of for adults to develop a food allergy. Data from a cross-sectional survey of adults living in the U.S. suggest at least 10.8% of adults are allergic to food. Information was gathered from October 2015 to September 2016.25

There were 40,443 adults who completed the survey and while 19% reported some type of food allergy, only 10.8% reported symptoms concurrent with an IgE reaction. The study was published in the Journal of the American Medical Association in a collaboration between Stanford food allergy expert Dr. Kari Nadeau and scientists at Northwestern University.26

The researchers believe this contradicts a long-held belief that most allergies develop in childhood. Past data had estimated 9% of adults had true food allergic responses. In the cohort, researchers found 38% of those they determined to have food allergies had experienced a reaction that sent them to the emergency room and 48% reported at least one food allergy was triggered after age 18.

Some food allergies developed as an adult can be severe, such as the reactions that graduate student Amy Barbuto experienced in a Thai restaurant. In an interview with a journalist from Texas Medical Center, Barbuto recounted her first allergic response at the restaurant.27

Before that day she had a food intolerance to gluten, but in 2011 she suffered an anaphylactic reaction when the wrong soy sauce was used in her food. Since then she was hospitalized 25 times from 2011 to 2020 for allergic reactions. She talked about the difficulty of avoiding gluten, saying:28

“It’s a hard one to avoid, even when you work your hardest to avoid it. My allergy is so severe that I could get exposed and not even know it. My food can look gluten-free, look normal … but all it would take is somebody having touched bread and then touched my plate.”

Reduce Your Potential Risk for Food Allergies

As Barbuto’s story demonstrates, food allergies can develop well into adulthood and become potentially lethal. There are steps you can take to lower your potential risk of developing an allergy or experiencing an anaphylactic response.

Your gut microbiome is vital to the health and optimal functioning of your immune system, which mediates an allergic response to food. By caring for your gut microbiome, you help protect your health. As I’ve written before in “How Your Gut Health Impacts Your Disease Risk”:

  • Fermented foods help repopulate your gut with healthy bacteria
  • Antacids change your stomach acid and negatively affect your gut microbiome
  • Steer clear of antibiotics, including in your food, unless absolutely necessary, and take a quality probiotic when you must use antibiotics
  • Reduce or eliminate processed foods, as they are high in sugar that feed harmful bacteria in your gut
  • Optimize your vitamin D levels through sensible sun exposure or supplementation if you live in areas with little sun. There are strategies you can use to achieve your vitamin D goals, as I shared in “The Most Important Paper Dr. Mercola Has Ever Written”


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00:22 , ,

If SARS-CoV-2 has frazzled your nerves, I have bad news for you. Scientists are already cooking up more virulent and lethal versions. In a January 22, 2021, Twitter post, biotech entrepreneur Yuri Deigin highlighted a study posted on the preprint server bioRxiv at the end of December 2020, saying:1

“Ok, the prize for the craziest and most dangerous gain-of-function research goes out to Italian virologists who took SARS[-CoV-]2 and passaged it in vitro in the presence of neutralizing antibodies.2 It quickly obliged and mutated to escape them. Yay for a novel, more dangerous SARS3!”

“Passaging” refers to a genetic engineering technique where a virus is grown in a series of different animal tissue cultures. With each “pass,” the virus will mutate slightly, gaining different functions.

Serial Passaging Allows Virus to Jump Species

As just one example, a potential outcome of this somewhat crude technique (considering the genetic engineering technology now available) would be that the virus could gain the ability to infect a host species it could not infect before. Some experts have speculated that this might be one way in which SARS-CoV-2 was created.

In an in-depth article3 published in New York magazine January 4, 2021, Nicholson Baker reviewed the history of viral gain-of-function research, providing the following example of serial passaging:

“Baric … described in this early paper how his lab was able to train a coronavirus, MHV, which causes hepatitis in mice, to jump species, so that it could reliably infect BHK (baby-hamster kidney) cell cultures.

They did it using serial passaging: repeatedly dosing a mixed solution of mouse cells and hamster cells with mouse-hepatitis virus, while each time decreasing the number of mouse cells and upping the concentration of hamster cells.

At first, predictably, the mouse-hepatitis virus couldn’t do much with the hamster cells, which were left almost free of infection, floating in their world of fetal-calf serum.

But by the end of the experiment, after dozens of passages through cell cultures, the virus had mutated: It had mastered the trick of parasitizing an unfamiliar rodent. A scourge of mice was transformed into a scourge of hamsters …”

Scientists Have Created Coronavirus That Escapes Antibodies

So, what exactly have they come up with now? As summarized by Deigin, researchers serial passaged live SARS-CoV-2 in plasma obtained from a recovered COVID-19 patient that had a high amount of neutralizing antibodies in it.4

For clarification, you have two types of antibodies. Neutralizing antibodies are, as the name implies, antibodies that neutralize (kill) viruses and prevent infection, whereas binding antibodies cannot prevent infection.

The neutralizing antibodies in the plasma successfully and completely neutralized the virus during the first seven passages, but then, the virus mutated to evade the antibodies. As explained by the authors:5

“The plasma fully neutralized the virus for 7 passages, but after 45 days, the deletion of F140 in the spike N-terminal domain (NTD) N3 loop led to partial breakthrough. At day 73, an E484K substitution in the receptor-binding domain (RBD) occurred, followed at day 80 by an insertion in the NTD N5 loop containing a new glycan sequon, which generated a variant completely resistant to plasma neutralization.”

In other words, they created a SARS-CoV-2 variant that bypasses acquired immunity and negates the immunity you normally would have after recovering from the infection. As such, it could be extremely lethal.

“Computational modeling predicts that the deletion and insertion in loops N3 and N5 prevent binding of neutralizing antibodies,” the authors say, adding:

“The recent emergence in the United Kingdom and South Africa of natural variants with similar changes suggests that SARS-CoV-2 has the potential to escape an effective immune response and that vaccines and antibodies able to control emerging variants should be developed.”

Selective Pressure of Vaccination May Pose a Problem

Now, further down in the paper, they point out that the reason they did this study was to determine “whether the authentic virus, under the selective pressure of the polyclonal immune response in convalescent or vaccinated people, can evolve to escape herd immunity and antibody treatment.”

Since the virus can mutate to evade neutralizing antibodies, then it could potentially mutate under the “selective pressure” of vaccination as well, which in turn raises the question: If we mass vaccinate, will we end up with a more lethal virus?

The solution these researchers seem to propose is to start thinking about vaccinating people for emerging SARS-CoV-2 variants, meaning we may need to develop a new vaccine — much like the seasonal flu vaccine, — to match the circulating strains of each season.

Considering the first COVID-19 mRNA vaccines (which are most accurately described as gene therapy) are wreaking absolute havoc on people’s health already, in many instances killing the patient within hours (although health officials adamantly claim their deaths have nothing to do with the vaccines), the idea of implementing a twice-a-year gene-therapy regimen against COVID-19 strikes me as assured destruction of the human race.

Is SARS-CoV-2 Result of Gain-of-Function Research in Wuhan?

Jamie Metzl is ageopolitics expert, World Health Organization adviser and senior fellow at the Atlantic Council. January 4, 2021, CBS News interviewed her about the “conspiracy theory” that SARS-CoV-2 was created in a biosecurity level 4 laboratory in Wuhan, China. Metzl believes the COVID-19 pandemic is the result of an accidental leak from that lab.

This, he says, is a logical conclusion based on the facts before us. First, Wuhan is far from the southern part of China where horseshoe bats (the supposed source host) exist.

Second, the Wuhan Institute of Virology (WIV) was known to have performed controversial gain-of-function research on bat coronaviruses and, according to U.S. diplomats who had visited the lab in 2018, significant safety shortcomings were apparent.6

Third, SARS-CoV-2’s closest relative (RaTG13) has been traced back to samples collected in 2012 from miners sickened after working in an abandoned mine in Mojiang. There’s no trace of the virus anywhere between 2012 and 2019, until it suddenly caused an outbreak in Wuhan.

Lastly, “We see this massive Chinese cover-up,” Metzl says, “destroying samples, shutting off access to databases, imprisoning journalists [and] silencing scientists.”

On top of that, Metzl points out that scientists working at the WIV have been unable to account for all the viruses in their database, and level 4 biosecurity laboratories around the world have experienced many safety breaches in the past.

Investigative Committees Are Severely Compromised

As noted by Metzl — who also recently published an op-ed about this in Newsweek — what we need is a full, independent, all-access forensic investigation into the origin of this virus. If we don’t, we will not be ready for whatever else that might be right around the corner.

He also warns that while the WHO has assembled a committee7 to investigate, China was granted veto power to decide who would be on that committee, and the primary investigation is to be carried out by Chinese representatives. The WHO’s committee will then simply review their findings. This questionable setup makes it highly unlikely that we’ll get to the truth.

Indeed, the members of the WHO’s investigative committee raises serious concerns about its ability to conduct an unbiased investigation. One of its members, Peter Daszak, Ph.D., is the president of EcoHealth Alliance, a nonprofit organization that has worked closely with the WIV.

When SARS-CoV-2 first emerged in Wuhan, the EcoHealth Alliance was actually providing funding to the WIV to collect and study novel bat coronaviruses. He has publicly and repeatedly dismissed the possibility of the pandemic being the result of a lab leak.8

Daszak Is the Fox Guarding the Hen House

Importantly, correspondence obtained by U.S. Right to Know (USRTK) show Daszak played a central role in the plot to obscure the lab origin of SARS-CoV-2 from the very beginning by crafting a scientific statement condemning such inquiries as “conspiracy theory.”9,10

This manufactured “consensus” was then relied on by the media to counter anyone presenting theories and evidence to the contrary. Daszak is also heading up a second commission to investigate the origin of the virus, The Lancet COVID-19 commission,11 thereby ensuring that the “consensus” will be maintained.

Ironically, in 2015, Daszak actually warned a global pandemic might occur from a laboratory incident and that “the risks were greater with the sort of virus manipulation research being carried out in Wuhan.”12 Earlier that year, he was also a key speaker at a National Academies of Science seminar on reducing risk from emerging infectious diseases.

Among the material Daszak presented at that meeting was a paper titled, “Assessing Coronavirus Threats,” which included an examination of the “spillover potential” from “genetic and experimental studies” on viruses. In particular, he highlighted the danger of experimenting on “humanized mice,” meaning lab mice that have been genetically altered to carry human genes, cells or tissues.

Considering Daszak’s personal involvement with gain-of-function research in general, and research efforts at WIV in particular, he has more than enough motivation to make sure the blame for the COVID-19 pandemic is not laid at the feet of researchers such as himself, especially those at WIV.

As long as he is part of these investigative committees, any conclusions they come up with will be suspect. In fact, according to reports, the WHO commission has no intention of investigating either the WIV13 or the lab escape theory!14

Links to US Commissioned Research

While most of the focus has been on the WIV, the U.S. and other Western nations are not without blame. In the video above, “The Next Revolution” host Steve Hilton reviews the origin of COVID-19, linking the outbreak to research around the world.

He starts reviewing research done by the Erasmus Centre in the Netherlands 10 years ago. There, they were able to get an influenza A/H5N1 virus to mutate and become airborne by injecting it into ferrets. This led to an explosion of gain-of-function virus research all around the world. Interestingly, that Dutch study was funded by none other than Dr. Anthony Fauci’s National Institute of Allergy and Infectious Diseases (NIAID).

While the original intent may have been noble — stay a step ahead of nature so we’re not surprised by natural mutations that might threaten the human population — by creating more virulent pathogens, the work itself ends up posing significant risk.

This was why, in 2014, the Obama administration put a moratorium on gain-of-function research after recent biosafety incidents had highlighted the risky nature of such study. The moratorium included pausing gain-of-function research on influenza, MERS and SARS viruses.

However, as noted by Hilton, Fauci has long been a steadfast advocate of this kind of research, and shortly before the moratorium was put into place, he had funded a project to assess the risk of bat coronavirus emergence and the “spillover potential at high-risk human-wildlife interfaces in China.” At the end of that project description, they state:

“Predictive models of host range (i.e. emergence potential) will be tested experimentally using reverse genetics, pseudovirus and receptor binding essays, and virus infection experiments across a range of cell cultures from different species and humanized mice.”

This is precisely the kind of research the Obama administration placed a moratorium on, but Fauci didn’t drop it. Instead, he contracted it out to the EcoHealth Alliance — the group run by Daszak. Daszak himself was the project leader. Over the next six years, EcoHealth Alliance received $3.75 million for projects relating to this investigation.

Fauci, Daszak and the WIV Appear To Be Key Culprits

Daszak, in turn, subcontracted out a key piece of the research — the gain-of-function part — to the WIV. In his report, Hilton reviews some of the papers published throughout this project, proving they were indeed part of the research Fauci funded.

He points out that while many admit the NIAID funded the WIV in general, a paper co-written by Daszak and Shi Zhengli, proves Fauci funded gain-of-function research on bat coronavirus specifically.

After Hilton’s team reached out to the NIH and Fauci for comment, the paper mysteriously disappeared. The paper in question, published in 2017, shows they built various chimeras based on bat coronaviruses collected. They then infected human cells with these chimeras in the lab, proving that their manmade viruses could replicate.

The genetic changes they made to these chimeras “unlocked a specific doorway to the human body,” Hilton explains, and this doorway is precisely the one SARS-CoV-2 uses, namely the ACE2 receptor.

While none of the genetically engineered viruses described in that 2017 paper is identical to SARS-CoV-2, the paper proves it’s possible to create these kinds of viruses using current technologies. What’s more, that project continued for another three years, which puts us into 2020. During those three years, any number of new variants may have been created.  

In light of the evidence, Fauci’s role as chief medical adviser to the White House and leader of the coronavirus task force is “completely untenable,” Hilton says. Indeed, his conflicts of interest make Fauci just as unsuitable for these roles as Daszak is for the ones he’s been assigned to.

They’re both involved up to their eyeballs in the research that may be the very source of this pandemic, yet both have been placed in key roles to inform, guide and direct the public on these matters. It’s scientific corruption at its finest.

Surely, there are other experts out there who would be just as, if not more, qualified for these roles. “Fauci must step aside until we get to the bottom of his role in creating — unintentionally, of course — this catastrophic global pandemic,” Hilton says. We also need to know whether the U.S. government is still funding research that could lead to another, even more devastating pandemic.



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